Product Name: ATAD3A Antibody
Species Reactivity: Human, Mouse, Rat
Tested Applications: ELISA, ICC, IF, WB
Applications: ATAD3A antibody can be used for detection of ATAD3A by Western blot at 1 μg/mL. Antibody can also be used for immunocytochemistry starting at 20 μg/mL. For immunofluorescence start at 20 μg/mL.
User Note: Optimal dilutions for each application to be determined by the researcher.
Predicted Molecular Weight:
Immunogen: ATAD3A antibody was raised against a 19 amino acid synthetic peptide near the amino terminus of human ATAD3A.The immunogen is located within amino acids 110 – 160 of ATAD3A.
Host Species: Rabbit
Purification: ATAD3A Antibody is affinity chromatography purified via peptide column.
Physical State: Liquid
CAS NO.: 24168-96-5
Product: Isoconazole (nitrate)
Buffer: ATAD3A Antibody is supplied in PBS containing 0.02% sodium azide.
Concentration: 1 mg/mL
Storage Conditions: ATAD3A antibody can be stored at 4˚C for three months and -20˚C, stable for up to one year. As with all antibodies care should be taken to avoid repeated freeze thaw cycles. Antibodies should not be exposed to prolonged high temperatures.
Clonality: Polyclonal
Conjugate: Unconjugated
Alternate Names: ATAD3A Antibody: ATPase family AAA domain-containing protein 3A
Accession NO.: Q9NVI7
Protein Ino: 283436220
Official Symbol: ATAD3A
Geneid: 55210
Background: ATAD3A Antibody: ATAD3A is a member of the AAA ATPase family, a family of proteins that catalyze ATP into ADP and are involved in several cellular functions such as cell-cycle regulation, protein proteolysis and transport. The AAA ATPase family is characterized by a highly conserved AAA motif containing Walker homology sequences and imparting ATPase activity. Mitochondrial membrane proteins ATAD3A/B contribute to the stabilization of nucleoids and may participate in the transformation pathway and the chemosensitivity of oligodendrogliomas. The gene encoding ATAD3A/B/C maps to human chromosome 1 and has been suggested to be an anti-apoptotic marker.
PubMed ID:http://aac.asm.org/content/53/6/2679.abstract
